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Director of DR. RAINA'S SAFE HANDS Sexologist Dr. Vinod Raina is the main one. He has had the privilege of working in the medical field for 22+ years as a top MBBS doctor. He also actively participates in and speaks at national and international medical conferences at Safdarjung Hospital. At national conferences, he has been provided. His excellent and enthusiastic approach to his work has helped more than many individuals. He frequently holds CMEs (continuing medical education) and seminars on the causes of HIV/AIDS, Pep Treatment and Sexual Dysfunction Treatment, and diet weight-reduction plan Nutrition to inform & refresh medical professionals about the job and improve his knowledge.
Inflicting tens of thousands of lives and sufferings, HIV has become a worldwide health emergency that affects all facets of society, regardless of gender, age, location, lifestyle, or nationality. Access Recently available modalities of treatment at various degrees, in addition to prevention, have changed the situation on a global scale. Our body's immune system protects us by identifying invasive pathogens (bacteria, fungi, viruses, and parasites) and providing an immediate reaction to those pathogens, preventing us from becoming unwell. T lymphocytes were eaten by immune system cells that are involved in the regulation and eradication of antigens. The CD4+ cells are the target of HIV while it is within the circulatory system.
the pollution occurred When HIV contamination first manifests, it is referred to as Primary Infection (HIV 1). Primary Infection (HIV 1) involves high viral load, which indicates the presence of several viruses, according to plasma or blood donation that might total more than one million millimeters. At this stage, the signs and symptoms are known as SERCONVERSION ILLNESS, and they include headache, sore throat, constant diarrheas, vomiting, nighttime discomfort, weight loss, and skin rashes. This SEROCONVERSION ILLNESS typically lasts for around 25 days. The amount of CD4 cells detected during Primary Infection (HIV 1) falls and may reach less than 200 cells/L. The HIV virus targets CD4+ cells in the lymph nodes and thymus, which makes the infected individual more vulnerable to opportunistic infections and lowers the thymus' capacity to produce T lymphocytes.
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